Research Library

Peptide profiles

Evidence-graded summaries of 11 peptide compounds — potential benefits, health considerations, study quality context, and direct links to the underlying research.

Disclaimer: These profiles are for informational purposes only and do not constitute medical advice. Evidence grades reflect the current state of published research. Preclinical evidence (animal/in vitro) cannot be directly extrapolated to humans. Always consult a qualified healthcare professional before using any peptide compound.

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Tissue Repair / GI
Preclinical
BPC-157
Body Protection Compound 157

Synthetic 15-amino-acid peptide derived from human gastric juice protein. Extensively studied in rodent models for wound healing, tendon repair, and GI mucosal protection. No completed human RCTs as of 2025.

Potential Benefits
  • Tendon and ligament healing in animal models
  • Gastric mucosal repair and GI tract integrity
  • Neuroprotective effects in CNS injury models
  • Anti-inflammatory via nitric oxide modulation
  • Promotes angiogenesis (VEGFR2 upregulation)
Considerations
  • Nearly all evidence is from rodent studies
  • No completed human randomized controlled trials
  • Theoretical pro-angiogenic concern with active cancer
  • Long-term human safety profile unknown
Recovery / Wound Healing
Early Human Data
TB-500
Thymosin Beta-4 Synthetic Analog

Synthetic version of the naturally occurring Thymosin Beta-4 protein present in nearly all nucleated human cells. Regulates actin dynamics, promotes cell migration and wound healing. Phase 2 human trials completed for cardiac and wound healing indications.

Potential Benefits
  • Accelerates wound healing via G-actin sequestration
  • Phase 2 data for cardiac repair post-myocardial infarction
  • Stem cell mobilization from bone marrow
  • Anti-fibrotic effects in liver and cardiac models
  • Promotes neurogenesis and neural cell survival
Considerations
  • Banned by WADA — significant risk for competitive athletes
  • Phase 2 cardiac trials showed modest efficacy; Phase 3 not done
  • Theoretical concern regarding angiogenesis in tumor contexts
  • Gray market TB-500 may differ from studied Tb4 protein
Skin / Anti-Aging
Early Human Data
GHK-Cu
Glycyl-L-Histidyl-L-Lysine Copper Complex

Naturally occurring copper-binding tripeptide that declines with age. Topical forms are widely used in cosmetics with supportive evidence. Injectable forms are a gray market research compound with limited human safety data.

Potential Benefits
  • Stimulates collagen and elastin synthesis
  • Accelerates wound healing and skin repair
  • Hair follicle stimulation in scalp studies
  • Broad anti-inflammatory gene expression profile
  • Antioxidant and free radical scavenging activity
Considerations
  • Most human evidence is topical cosmetic data
  • Injectable human safety profile understudied
  • Copper overload theoretically possible at high doses
  • Gene expression claims based largely on bioinformatics
Cognitive / Neuroprotective
Human Clinical Data
Semax
ACTH(4–7)–Pro–Gly–Pro Heptapeptide

ACTH-derived heptapeptide approved as a pharmaceutical in Russia and Ukraine. Upregulates BDNF and NGF, modulates dopaminergic and serotonergic systems. The most clinically studied neuropeptide on this list outside of Russia.

Potential Benefits
  • Approved in Russia for ischemic stroke recovery
  • Upregulates BDNF and NGF for neural plasticity
  • Cognitive enhancement in healthy and clinical populations
  • Anxiolytic effects at moderate doses
  • Rapid intranasal delivery
Considerations
  • Clinical trials primarily from Russia; limited Western replication
  • Can cause irritability or anxiety at higher doses
  • Intranasal bioavailability and dosing consistency varies
  • Drug interactions not well characterized
Cognitive / Anxiolytic
Human Clinical Data
Selank
Thr-Lys-Pro-Arg-Pro-Gly-Pro (Tuftsin Analog)

Synthetic analog of the immunopeptide tuftsin, registered as a pharmaceutical in Russia for anxiety and as a nootropic. Modulates GABA-A sensitivity, serotonin, and dopamine. Anxiolytic without the dependence risk of benzodiazepines in animal models.

Potential Benefits
  • Approved in Russia for generalized anxiety disorder
  • Anxiolytic without sedation or physical dependence
  • Nootropic — improved working memory and focus
  • Immunomodulatory: enhances T-cell and macrophage activity
  • Modulates enkephalin degradation
Considerations
  • Human trials primarily from Russia; limited independent replication
  • Some users report lethargy, especially intranasally
  • Drug interaction profile with CNS medications understudied
  • Long-term neurochemical effects unknown
Metabolic / Weight Loss
Phase 2 RCT
Retatrutide
LY3437943 — Triple GIP/GLP-1/Glucagon Agonist

Eli Lilly investigational peptide simultaneously activating GIP, GLP-1, and glucagon receptors. Phase 2 NEJM results (2023) showed up to 24.2% body weight reduction at 48 weeks. Phase 3 trials ongoing as of 2025.

Potential Benefits
  • Up to 24.2% body weight loss in Phase 2 (48 weeks)
  • Triple receptor mechanism: GIP + GLP-1 + glucagon
  • Improved glycemic control and insulin sensitivity
  • Once-weekly subcutaneous injection
  • Potentially superior efficacy to semaglutide and tirzepatide
Considerations
  • GI adverse effects (nausea, vomiting) common — class effect
  • Not yet FDA approved; Phase 3 safety data pending
  • Rebound weight gain upon discontinuation expected
  • Long-term cardiovascular outcomes trial not complete
  • Gray market versions pose severe quality and safety risk
Metabolic / Body Composition
FDA Approved
Tesamorelin
GHRH(1–44) Analog (Egrifta SV)

FDA-approved GHRH analog (Egrifta, 2010) for HIV-associated lipodystrophy. Stimulates physiologic GH release from the pituitary. Research interest has expanded to NAFLD reduction and cognitive function in aging adults.

Potential Benefits
  • FDA-approved: reduces visceral fat in HIV lipodystrophy
  • Physiologic GH stimulation via pituitary (not direct GH)
  • Improves cognitive function in older adults (clinical trial data)
  • Reduces liver fat in NAFLD patients
  • Lower IGF-1 spike risk vs. exogenous GH injection
Considerations
  • FDA approval limited to HIV lipodystrophy; other uses are off-label
  • Fluid retention, joint pain, carpal tunnel reported
  • Can impair glucose metabolism in diabetics
  • Expensive through legitimate channels
GH Secretagogue / Recovery
Early Human Data
Ipamorelin
Selective Ghrelin Mimetic Pentapeptide

Selective GH secretagogue that stimulates pulsatile GH release without meaningfully elevating cortisol or prolactin. Phase 2 data for postoperative bowel function. Widely used off-label for recovery and body composition.

Potential Benefits
  • Selective GH release without cortisol/prolactin elevation
  • Phase 2 human data for postoperative GI motility
  • Improved body composition in early human and animal studies
  • Short half-life mimics physiologic GH pulsatility
  • Commonly stacked with GHRH analogs for synergistic effect
Considerations
  • Limited high-quality human RCT data for anti-aging endpoints
  • Water retention and headache at higher doses
  • Chronic use: long-term GH axis suppression risk
  • Banned in sport by WADA (S2)
  • Not FDA approved for any indication
GH Secretagogue / Recovery
Early Human Data
CJC-1295
GHRH(1–29) with Drug Affinity Complex (DAC)

Long-acting GHRH analog with a DAC modification extending half-life to 6–8 days. Phase 1/2 human data published in 2006 demonstrates dose-dependent sustained GH and IGF-1 elevation. Creates sustained rather than pulsatile GH release.

Potential Benefits
  • Phase 1/2 human data: dose-dependent GH/IGF-1 elevation
  • Extended dosing interval (1–2x weekly)
  • Lean mass and fat loss via GH axis stimulation
  • Synergistic with GH secretagogues like ipamorelin
  • No direct cortisol or prolactin elevation
Considerations
  • Sustained GH elevation differs from physiologic pulsatile signaling
  • Long-term IGF-1 elevation: theoretical cancer risk with chronic use
  • Water retention, carpal tunnel, joint pain reported
  • DAC modification technically complex — gray market quality varies
  • Banned in sport (WADA S2)
Longevity / Metabolic
Preclinical
MOTS-c
Mitochondrial ORF of the Twelve S rRNA type-c

Mitochondria-encoded 16-amino-acid peptide discovered in 2015. Activates AMPK, improves insulin sensitivity, and counteracts metabolic aging. Circulating levels decline with age and increase with exercise. Human trials in very early stages.

Potential Benefits
  • AMPK activation — central energy metabolism regulator
  • Improved insulin sensitivity in aging and obese animal models
  • Exercise mimetic: muscle function improvement without exercise
  • Anti-obesity effects in high-fat diet rodent models
  • Endogenous levels correlate with longevity in centenarian studies
Considerations
  • Nearly all evidence is from mouse/rat models
  • Human PK and bioavailability largely unknown
  • No completed human clinical trials for any indication
  • 2015 discovery means very limited longitudinal data
  • Extremely early-stage — highest uncertainty on this list
Metabolic / Fat Loss
Phase 2/3 Data
AOD-9604
hGH C-Terminal Fragment (Residues 177–191)

C-terminal fragment of human growth hormone that stimulates fat oxidation and lipolysis without IGF-1-mediated effects. Multiple Phase 2 trials conducted; Phase 3 failed to meet primary obesity endpoints. Development discontinued.

Potential Benefits
  • Stimulates fat oxidation without IGF-1-mediated effects
  • Does not cause glucose intolerance unlike exogenous GH
  • Phase 2 data showed favorable fat loss vs. placebo
  • Investigated for cartilage regeneration in osteoarthritis
  • Previously TGA-considered as food ingredient in Australia
Considerations
  • Phase 3 obesity trial failed to meet primary endpoints
  • Commercial drug development discontinued after Phase 3 failure
  • Modest efficacy signals vs. lifestyle intervention
  • Headache, edema, injection site reactions reported
  • Gray market purity and dosing accuracy concerns
Anti-Inflammatory / GI
Preclinical
KPV
Lys-Pro-Val — C-terminal α-MSH Tripeptide

C-terminal tripeptide of alpha-melanocyte-stimulating hormone retaining core anti-inflammatory activity via MC1R binding and NF-κB inhibition. Research focus on IBD, skin inflammation, and oral nanoparticle delivery for GI bioavailability.

Potential Benefits
  • Potent NF-κB inhibition — central inflammatory pathway
  • Reduces intestinal inflammation in colitis animal models
  • Anti-inflammatory in skin via MC1R
  • Accelerated wound closure in animal models
  • Oral nanoparticle delivery system under development for GI delivery
Considerations
  • All significant evidence is preclinical (rodent/cell models)
  • No published human clinical trial data
  • Negligible oral bioavailability without nanoparticle delivery
  • MC1R agonism: possible pigmentation changes at high doses
  • Extremely limited human safety or PK data